Welcome to MolDiag-PaCa
[1] LÜTTGES, J., GALEHDARI, H., BRÖCKER, V., SCHWARTE-WALDHOFF, I., HENNE-BRUNS, D., KLÖPPEL, G., SCHMIEGEL, W., HAHN, S.A.: Allelic loss is often the first hit in the biallelic inactivation of the p53 and DPC4 genes during pancreatic carcinogenesis. Am. J. Pathol. 158: 1677-1683 (2001)

[2] SIPOS, B., KLAPPER, W., KRUSE, M.-L., KALTHOFF, H., KERJASCHKI, D., KLÖPPEL, G.: Expression of lymphangiogenic factors and evidence of intratumoral lymphangiogenesis in pancreatic endocrine tumors. Am J Pathol 165: 1187-1197 (2004)

[3] GRÜTZMANN, R., PILARSKY, C., AMMERPOHL, O., LÜTTGES, J., BÖHME, A., SIPOS, B., FOERDER, M., ALLDINGER, I., JAHNKE, B., SCHACKERT, H.K., KALTHOFF, H., KREMER, B., KLÖPPEL, G., SAEGER, H.D.: Gene expression profiling of microdissected pancreatic ductal carcinomas using high-density DNA microarrays. Neoplasia 6: 611-622 (2004)

[4] MÜERKÖSTER, S., WEGEHENKEL, K., ARLT, A., WITT, M., SIPOS, B., KRUSE, M.L., SEBENS, T., KLÖPPEL, G., KALTHOFF, H., FÖLSCH, U.R., SCHÄFER, H.: Tumor stroma interactions induce chemoresistance in pancreatic ductal carcinoma cells involving increased secretion and paracrine effects of nitric oxide and interleukin-1β. Cancer Res 64: 1331-1337 (2004)

[5] KLÖPPEL, G., LÜTTGES, J.: The pathology of ductal-type pancreatic carcinomas and pancreatic intraepithelial neoplasia: insights for clinicians. Curr Gastroenterol Rep 6: 111-118 (2004)